| Erhard Bieberich |
Rotations 2,3,4 |
Extracellular vesicles in Alzheimer's |
| Cheavar Blair |
Rotations 1,2,3,4 |
Our lab utilizes stem cell derived cardiomyocytes to understand sarcomere regulation. |
| Ken Campbell |
Rotations 1,2,3,4 |
Translational research that seeks better therapies for patients who have heart failure. The lab integrates biophysics, biochemistry, biobanking, and computer modeling. |
| Alan Daugherty |
Rotations 1,2,3,4 |
Vascular pathology with emphasis on aortic diseases and atherosclerosis |
| Brian Delisle |
Rotations 1,2,4 |
We study how environment and behavior impact cardiac physiology at the molecular, cellular, and organism level. |
| Gregory Frolenkov |
Rotations 1,2,3 |
Molecular mechanisms of mechanosensitivity and hearing loss. |
| Christopher Fry |
Rotations 1,2,3,4 |
We elucidate the cellular and molecular mechanisms underlying skeletal muscle adaptation to promote tissue health and function to enhance recovery after injury and during illness. |
| Nate Helsley |
Rotations 3,4 |
The Helsley lab aims to identify mechanisms linking dietary nutrient metabolism to the development of obesity-associated disorders, including diabetes, liver cancer, and cardiovascular disease. |
| Brad Hubbard |
Rotations 1,2,4 |
The research interests of the Hubbard lab are understanding the pathobiology of traumatic brain injury (TBI) with a focus on blood-brain barrier and mitochondrial disease mechanisms. |
| Lance Johnson |
Rotations 1,2,3 |
We combine in vitro, in vivo, and clinical research systems to study Apolipoprotein E (APOE), the strongest genetic risk factor for Alzheimer's disease |
| Shannon Macauley |
Rotations 1,2,3,4 |
The main focus of my research is to understand how metabolic dysfunction affects sleep, neuroinflammation, and vascular function in the setting of Alzheimer's disease (AD). Ultimately, the goal is to leverage these findings as therapeutic targets for treating CNS diseases. |
| Allison Owen |
Rotations 1,2,3,4 |
We investigate mechanisms of skeletal muscle dysfunction resultant of critical illnesses (burn trauma and sepsis), with a special interest in immunoregulatory factors. |
| Samir Patel |
Rotations 1,2,3,4 |
Characterizing rodent model of sepsis survivor following spinal trauma and targeting mitochondrial dysfunction to treat spinal injury |
| Hiroshi Saito |
Rotations 2,3,4 |
Understanding the mechanisms and developing treatments for long-term physical and cognitive dysfunction after sepsis. |
| Jonathan Satin |
Rotations 1,2,3 |
The Rad lab is focused on mechanisms of heart function with the long-term goal to develop the basis for new therapeutics to treat heart failure. |
| A. Catalina Velez-Ortega |
Rotations 1,2,3,4 |
Molecular machinery involved in the activity-dependent plasticity of the cytoskeleton in inner ear sensory organelles, and mechanisms of protection against noise-induced hearing loss. |